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Both X4 and R5 HIV are present in the seminal fluid , which enables the virus to be transmitted from a male to his sexual partner. Penny is puzzled and laughs. This is one of the most densely glycosylated molecules known and the density is sufficiently high to prevent the normal maturation process of glycans during biogenesis in the endoplasmic and Golgi apparatus. These hosts have adapted to the presence of the virus, [90] which is present at high levels in the host's blood, but evokes only a mild immune response, [91] does not cause the development of simian AIDS, [92] and does not undergo the extensive mutation and recombination typical of HIV infection in humans.

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Both X4 and R5 HIV are present in the seminal fluid , which enables the virus to be transmitted from a male to his sexual partner. Penny is puzzled and laughs. This is one of the most densely glycosylated molecules known and the density is sufficiently high to prevent the normal maturation process of glycans during biogenesis in the endoplasmic and Golgi apparatus. These hosts have adapted to the presence of the virus, [90] which is present at high levels in the host's blood, but evokes only a mild immune response, [91] does not cause the development of simian AIDS, [92] and does not undergo the extensive mutation and recombination typical of HIV infection in humans.

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  1. The six remaining genes, tat, rev, nef, vif, vpr, and vpu or vpx in the case of HIV-2 , are regulatory genes for proteins that control the ability of HIV to infect cells, produce new copies of virus replicate , or cause disease. That gives Joanna an idea.

  2. In the tonsils and adenoids of HIV-infected patients, macrophages fuse into multinucleated giant cells that produce huge amounts of virus. A number of studies with subtype B-infected individuals have determined that between 40 and 50 percent of AIDS patients can harbour viruses of the SI and, it is presumed, the X4 phenotypes.

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